CameronJayBauer
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Honestly, I don't think anyone can truthfully say how long is the useful life with any sort of realistic accuracy. You'll encounter the typical "six months replacement" regurgitation, but I don't actually think this is based on any empirical evidence whatsoever. There are two main problems that would hinder anyone to give an accurate answer: very few actually have the appropriate equipment to realistically measure their output (I've seen a wonderful piece of equipment and for some reason I can't even get the manufacturer to give me a damned quote on what it costs), and no one really knows how much UVB is essential to not only promote good health, but ensure it.
Now, having said this, I have some anecdotal evidence that, contrary to what is often said (even by myself), seems to point in a direction many would argue against (including myself). Years ago when dinosaurs roamed North America and I was just starting out my first steps in research with tegus, my supervisor had brought me my initial collection of something like 35 baby tegus and had me set them up to his guidelines. Now, I was no where near as knowledgeable (or confident) as I am now, but even them I was "well, I wouldn't set them up that way, but you're the boss". Basically, they were kept in large fiberglass bins with brooder heat lamps (the big red bulbs), being fed a ground meat mash with chopped veggies and vitamin/mineral supplementation. It wasn't too long before some started showing the early signs of calcium deficiency (muscle tremors). Called in our departmental veterinarian, he concurred, we gave them some shots of calcium (and I think vit D3), tremors went away. Then came back. So we tried mixing up the shots, including other vitamins and such associated with the whole calcium regulation pathways, each time succeeding in driving off the tremors only to have them return later. We really analysed what I was feeding them and were quite certain that mineral and nutritional needs were being met. And the numbers of tegus affected kept growing. Being somewhat analytical and wanting to figure out what was going wrong, I started dividing up the tegus according to the severity/repeatedness I was observing. Before long, had run out of suitable housing in our lab, and so did what I usually did: brought in my own stuff to completely separate all the affected animals. Set them up individually, and before long, started to see a pattern. Some of the tegus kept developing tremors, some got better and we never saw tremors in them again. Looking at who got treated with what, there was no clear pattern to indicate which treatment we'd tried seemed to be working. Until it dawned on me - the ones that were no longer exhibiting tremors were ones now being housed in MY terrariums with MY lighting.....fluorescent bulbs with UV output. Here's the kicker: this was BEFORE the selection of bulbs we have now, and I certainly didn't bring in brand new bulbs at my own expense, these were old Vitalite bulbs, some before the "patented twist" line, that I had kicking around, wasn't using anymore as I thought they were likely "spent". Some of these bulbs were likely 5-7 years old, and the Vitalites never had the intensity we now have in reptile lighting. These old bulbs clearly had enough UVB to correct the problem.
Here's the issue, though. Even with this story behind me, this is NOT a statement that any old bulb will do, or that old bulbs will continue to produce adequate UVB for years. Only that in this situation, it was enough to adjust a "bad" situation and make it better. Since then I've learned a lot more and understand the situation better than it appears most do. Most of us are aware that UVB helps the calcium metabolism pathway. Some understand that what UVB does is convert pre-vitamin D3 to bioactive pro-vitaminD3 (it's far more complicated than this, but this is suitable for what we're talking about). Fewer seem to understand that vitamin D3 is a lipid soluble vitamin (unlike most), and therefore can be sequestered and stored in tissues, and as such can thereby reach hazardous, toxic levels (water soluble vitamins we just pee out). This is because surprisingly we don't have an innate, biological method of regulating our vitamin D3 content. What very few seem to realise (including the human health practitioners that go on about how many units of vitamin D3 you should intake every day) is that once the vitamin D3 levels reach a sufficient concentration in our plasma and with sufficient intensity of UVB, that the pro-vitamin D3 is FURTHER converted to two inert forms that are apparently non-toxic at any concentration. So we need some UVB to photoactivate vitamin D3 into a usable form, and then we need MORE UVB of sufficient intensity to properly regulate Vitamin D3 levels.
Short'n'sweet: A suitable fluorescent bulb can likely produce UVB of sufficient intensity to activate some vitamin D3 for quite some time, but to produce enough UVB to properly regulate the vitamin D3 ........... no one really knows what intensity is necessary or how long the various bulbs can maintain that intensity.
I seriously love your background knowledge of tegu research. This is an awesome bit of information. Regardless if we don't have a concrete conclusion on the amount of UVB necessary, this alone gives me a far better understanding of the role UVB lighting has, thank you.
Do you have any opinions regarding an unhealthy amount of UVB exposure?